Scottish TSE Network November Symposium Announcement Event: 12 November 2012

Chair: Prof Hugh Perry, University of Southampton, Southampton UK

Location: The Roslin Institute Building Auditorium

If you would like to book a place at this event, please let Gila Holliman know.

Cost: £125.



Title: Is Alzheimer’s Disease a transmissible disease?



Speakers:

Session 1:

Prof Bob Will, National CJD Surveillance Unit, Edinburgh UK

Prof James Ironside, National CJD Surveillance Unit, Edinburgh UK

Prof Lary Walker, Emory School of Medicine, Atlanta USA



Session 2:

Prof Mathias Jucker, Hertie Institute for Clinical Brain Research, Stuttgart Germany

Prof William Van Nostrand, Stony Brook University, Stony Brook USA

Dr Claudio Soto, University of Texas Medical School, Houston USA



Session 3:

Dr Fabrizio Tagliavini, Instituto Neurologico Carlo Besta, Milan Italy

Prof Pedro Piccardo, Food and Drug Administration, Washington DC USA

Dr Bruce Chesebro, National Institutes of Health, Missoula USA





http://www.roslin.ed.ac.uk/events/20...-announcement/









From: Terry S. Singeltary Sr.
Sent: Wednesday, May 16, 2012 3:29 PM
To: BSE-L@LISTS.AEGEE.ORG
Subject: [BSE-L] Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?

Proposal ID: 29403

Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?

Background

Alzheimer’s disease and Transmissible Spongiform Encephalopathy disease have both been around a long time, and was discovered in or around the same time frame, early 1900’s. Both disease, and it’s variants, in many cases are merely names of the people that first discovered them. Both diseases are incurable and debilitating brain disease, that are in the end, 100% fatal, with the incubation/clinical period of the Alzheimer’s disease being longer than the TSE prion disease. Symptoms are very similar, and pathology is very similar. I propose that Alzheimer’s is a TSE disease of low dose, slow, and long incubation disease, and that Alzheimer’s is Transmissible, and is a threat to the public via the many Iatrogenic routes and sources. It was said long ago that the only thing that disputes this, is Alzheimer’s disease transmissibility, or the lack of. today, there is enough documented science (some confidential), that shows that indeed Alzheimer’s is transmissible. The risk factor for friendly fire, and or the pass-it-forward mode i.e. Iatrogenic transmission is a real threat, and one that needs to be addressed immediately.

Methods

Through years of research, as a layperson, of peer review journals, transmission studies, and observations of loved ones and friends that have died from both Alzheimer’s and the TSE prion disease i.e. Heidenhain Variant Creutzfelt Jakob Disease CJD.

Results

The likelihood of many victims of Alzheimer’s disease from the many different Iatrogenic routes and modes of transmission as with the TSE prion disease. TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit. you cannot cook the TSE prion disease out of meat. you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE. Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well. the TSE prion agent also survives Simulated Wastewater Treatment Processes. IN fact, you should also know that the TSE Prion agent will survive in the environment for years, if not decades. you can bury it and it will not go away. TSE prion agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area. it’s not your ordinary pathogen you can just cook it out and be done with. that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent.

Conclusions

There should be a Global Congressional Science round table event (one of scientist and doctors et al only, NO CORPORATE, POLITICIANS ALLOWED) set up immediately to address these concerns from the many potential routes and sources of the TSE prion disease, including Alzheimer’s disease, and a emergency global doctrine put into effect to help combat the spread of Alzheimer’s disease via the medical, surgical, dental, tissue, and blood arena’s. All human and animal TSE prion disease, including Alzheimer’s should be made reportable in every state, and Internationally, WITH NO age restrictions. Until a proven method of decontamination and autoclaving is proven, and put forth in use universally, in all hospitals and medical, surgical arena’s, or the TSE prion agent will continue to spread. IF we wait until science and corporate politicians wait until politics let science _prove_ this once and for all, and set forth regulations there from, we will all be exposed to the TSE Prion agents, if that has not happened already. what’s the use of science progressing human life to the century mark, if your brain does not work?

http://aaic.scsubmissions.com/index.aspx





combined cannot exceed 350 Words



shortened to proper word count ;

Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?


Background

Alzheimer’s disease and Transmissible Spongiform Encephalopathy disease have both been around a long time, and was discovered in or around the same time frame, early 1900’s. Both diseases are incurable and debilitating brain disease, that are in the end, 100% fatal, with the incubation/clinical period of the Alzheimer’s disease being longer (most of the time) than the TSE prion disease. Symptoms are very similar, and pathology is very similar.

Methods

Through years of research, as a layperson, of peer review journals, transmission studies, and observations of loved ones and friends that have died from both Alzheimer’s and the TSE prion disease i.e. Heidenhain Variant Creutzfelt Jakob Disease CJD.

Results

I propose that Alzheimer’s is a TSE disease of low dose, slow, and long incubation disease, and that Alzheimer’s is Transmissible, and is a threat to the public via the many Iatrogenic routes and sources. It was said long ago that the only thing that disputes this, is Alzheimer’s disease transmissibility, or the lack of. The likelihood of many victims of Alzheimer’s disease from the many different Iatrogenic routes and modes of transmission as with the TSE prion disease.

Conclusions

There should be a Global Congressional Science round table event set up immediately to address these concerns from the many potential routes and sources of the TSE prion disease, including Alzheimer’s disease, and a emergency global doctrine put into effect to help combat the spread of Alzheimer’s disease via the medical, surgical, dental, tissue, and blood arena’s. All human and animal TSE prion disease, including Alzheimer’s should be made reportable in every state, and Internationally, WITH NO age restrictions. Until a proven method of decontamination and autoclaving is proven, and put forth in use universally, in all hospitals and medical, surgical arena’s, or the TSE prion agent will continue to spread. IF we wait until science and corporate politicians wait until politics lets science _prove_ this once and for all, and set forth regulations there from, we will all be exposed to the TSE Prion agents, if that has not happened already.

end...tss


Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?

source references

Ann N Y Acad Sci. 1982;396:131-43.

Alzheimer's disease and transmissible virus dementia (Creutzfeldt-Jakob disease).

Brown P, Salazar AM, Gibbs CJ Jr, Gajdusek DC.

Abstract

Ample justification exists on clinical, pathologic, and biologic grounds for considering a similar pathogenesis for AD and the spongiform virus encephalopathies. However, the crux of the comparison rests squarely on results of attempts to transmit AD to experimental animals, and these results have not as yet validated a common etiology. Investigations of the biologic similarities between AD and the spongiform virus encephalopathies proceed in several laboratories, and our own observation of inoculated animals will be continued in the hope that incubation periods for AD may be even longer than those of CJD.



http://onlinelibrary.wiley.com/doi/1...849.x/abstract




BSE101/1 0136


IN CONFIDENCE

CMO

From: Dr J S Metters DCMO

4 November 1992


TRANSMISSION OF ALZHEIMER TYPE PLAQUES TO PRIMATES



http://collections.europarchive.org/...1/04001001.pdf





CJD1/9 0185

Ref: 1M51A

IN STRICT CONFIDENCE

From: Dr. A Wight Date: 5 January 1993

Copies:

Dr Metters

Dr Skinner

Dr Pickles

Dr Morris

Mr Murray

TRANSMISSION OF ALZHEIMER-TYPE PLAQUES TO PRIMATES



http://collections.europarchive.org/...1/05004001.pdf




Wednesday, January 5, 2011

ENLARGING SPECTRUM OF PRION-LIKE DISEASES Prusiner Colby et al 2011 Prions

David W. Colby1,* and Stanley B. Prusiner1,2



http://cshperspectives.cshlp.org/con....full.pdf+html




http://betaamyloidcjd.blogspot.com/2...rion-like.html


Tuesday, October 4, 2011



Molecular Psychiatry

advance online publication 4 October 2011; doi: 10.1038/mp.2011.120

De novo induction of amyloid-ß deposition in vivo

Our results suggest that some of the typical brain abnormalities associated with AD can be induced by a prion-like mechanism of disease transmission through propagation of protein misfolding. These findings may have broad implications for understanding the molecular mechanisms responsible for the initiation of AD, and may contribute to the development of new strategies for disease prevention and intervention. Keywords: amyloid; prion; protein misfolding; disease transmission



http://www.nature.com/mp/journal/vao...p2011120a.html



see more here ;


http://betaamyloidcjd.blogspot.com/2...eposition.html




http://transmissiblespongiformenceph...smissible.html




Wednesday, September 21, 2011


PrioNet Canada researchers in Vancouver confirm prion-like properties in Amyotrophic Lateral Sclerosis (ALS)



http://transmissiblespongiformenceph...vancouver.html




http://betaamyloidcjd.blogspot.com/




snip...end


Thank You for accepting my submission

# 29403, Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ? and the opportunity to present it, at the Alzheimer’s Association International Conference 2012 (AAIC), as a poster presentation. However, with great sadness, I must regretfully decline the invitation due to a medical reasons, and traveling to Canada, of which is not possible. ...


Thank You,

With Kindest Regards,

I am sincerely,

Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518

flounder9@verizon.net


From:

Sent: Saturday, April 07, 2012 8:20 PM

To: Terry S. Singeltary Sr.

Subject: RE: re-submission

Dear Terry,

Yes, your proposal was accepted as a poster presentation. Please decline the invitation if appropriate.

Best Regards,

______________________________________

Alzheimer’s Association – National Office
225 North Michigan Avenue – Floor 17
Chicago, Illinois 60601


=============snip...end...source reference...# 29403==========




http://betaamyloidcjd.blogspot.com/2...-error-of.html




Wednesday, May 16, 2012


Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?


Proposal ID: 29403




http://betaamyloidcjd.blogspot.com/2...smissible.html




TSS


==============JUNE 2012 UPDATE TSE USA=====================



Wednesday, June 27, 2012

First US BSE Case Since 2006 Underscores Need for Vigilance

Neurology Today 21 June 2012


http://transmissiblespongiformenceph...ince-2006.html




Tuesday, June 26, 2012

Creutzfeldt Jakob Disease Human TSE report update North America, Canada, Mexico, and USDA PRION UNIT as of May 18, 2012

type determination pending Creutzfeldt Jakob Disease (tdpCJD), is on the rise in Canada and the USA

http://creutzfeldt-jakob-disease.blo...human-tse.html





MOM DOD 12/14/97 CONFIRMED HVCJD I.E. THE HEIDENHAIN VARIANT OF CREUTZFELDT JAKOB DISEASE...TSS



Tuesday, July 29, 2008

Heidenhain Variant Creutzfeldt Jakob Disease Case Report

FINAL AUTOPSY DIAGNOSIS

I. Brain: Creutzfeldt-Jakob disease, Heidenhain variant.


SKROLL down a bit for Mom's autopsy of hvCJD. ...


http://creutzfeldt-jakob-disease.blo...ldt-jakob.html




mom, I’m still here damn’t. ...




TSS




LAYPERSON




Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
flounder9@verizon.net